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While the oral bioavailability of aspirin is 40-50%, clinical studies indicate that the majority of aspirin's irreversible inhibition of platelet COX-1 takes place in the presystemic (portal) circulation, resulting in aspirin's antiplatelet effect being largely independent of systemic bioavailability (Pedersen & FitzGerald, 1984

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References Mayer, E

Acute inflammatory response following increased blood-brain barrier permeability induced by focused ultrasound is dependent on microbubble dose