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The release of pro-inflammatory cytokines such as IL-1, IL-6, TNF-, and IL-17 leads to increased immune-cell activation and recruitment, creating an environment conducive to viral replication [2]

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Lin et al., 2022) This oxidative dysregulation is thought to exacerbate neuroinflammation, mitochondrial dysfunction, and synaptic abnormalities, which may underlie some of the core behavioral and cognitive impairments seen in ASD (Gevezova et al., 2023)

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