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Thrombosis in extracorporeal membrane oxygenation (ECMO) circuits

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Mechanistically, CRAT overexpression markedly induced G1-S cell cycle arrest and apoptosis of OC cells, which was supported by Ki-67 IHC and TUNEL staining assays, showing fewer proliferating and more apoptotic cells in xenograft tumors developed from CRAT overexpressing HEY cells