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Conclusion Increasing evidence suggests that anti-diabetes medicine, GLP-1R agonists, have multiple neuroprotective mechanisms in AD models, such as anti-inflammation, anti-oxidative stress, reducing A aggregation/deposition and tau protein hyperphosphorylation, reducing neuronal apoptosis and neurotoxicity, increasing cell proliferation and neurogenesis, increasing synaptic plasticity, and other beneficial effects
The combination of GLP-1-mediated insulin effects, keto-induced glucose reduction, and any other diabetes medications creates real hypoglycemia risk that needs active management
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doi: 10.1111/cbdd.14586 63
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