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Cytokine profiling revealed that only HA + GSH-C4 elicited significant modulation of tear mediators, and post-treatment cytokine levels correlated directly with other clinical metrics, consistent with a glutathione-mediated rebalancing of the ocular surface microenvironment

However, the stability of C23pAcF increases after the disulfide bond is destroyed, which is the opposite of the stability change of C96pAcF

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