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When activated by peripheral infection, systemic inflammation, or oxidative stress, microglia release a barrage of inflammatory cytokines that disrupt neuronal communication and damage brain tissue

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In terms of GLP1related mechanisms that might affect vulnerability to or development of AUD, a genetic study composed of four human genetic association studies has shown that the GLP1 receptor 168Ser allele is associated with increased alcohol selfadministration and a higher fMRI bloodoxygenleveldependent (BOLD) response in the globus pallidus when receiving rewarding feedback in the monetary incentive delay task

Early-stage studies are exploring how this multi-receptor activity influences metabolic signalling compared to dual or single agonists