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Postbiotics are considered more promising than probiotics as they elicit similar effects, despite being inactivated, thus relegating concerns of strain activity and stability that have been raised about probiotics

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The above point (2) provides evidence that loss of GLP-1 secretion with immediate pathophysiological consequences (reduced incretin effect as part of impaired postprandial beta cell function) calls for a replacement therapy with incretin-based glucose-lowering medications [3]