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A search of the literature shows numerous other investigations regarding the role of the eNOS enzyme and NO itself, which finally suggest that NO in moderate amounts can be neuroprotective while NO in large concentrations will be neurotoxic or cytotoxic

MTHFR gene mutations Certain MTHFR variants can reduce the enzymes activity so much that homocysteine cant be efficiently recycled into methionine, potentially leading to lower baseline homocysteine levels in some individuals, particularly when combined with low B-vitamin intake

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