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One likely explanation is that endocrine therapy creates a metabolic environment characterized by estrogen deficiency, insulin resistance, and inflammation that diminishes the pharmacological effects of GLP-1RAs on weight loss and glucose metabolism in BC patients [20]

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The research conversation around GHK-Cu has included reports of effects on tissue repair, on collagen and extracellular matrix gene expression, on antioxidant gene networks, on hair follicle biology, and on broader gene expression patterns characterized in transcriptomic studies